Hyperlipidemia and Atherosclerosis.
نویسندگان
چکیده
منابع مشابه
Postprandial hyperlipidemia and atherosclerosis.
The development of the remnant like particle (RLP) method for conveniently measuring serum remnant lipoprotein levels in 1993 promoted much research on atherogenic significance and metabolism of remnant lipoproteins. This research brought about many results as the following. A novel apolipoprotein B48 receptor incorporating remnant lipoproteins into macrophages in arterial wall was discovered a...
متن کاملMouse models of hyperlipidemia and atherosclerosis.
The mouse is the most utilized model to study lipids and atherosclerosis. Before the advent of the techniques of genetic manipulation it was well known that different inbred strains had varying degrees of susceptibility to diet-induced atherosclerosis. The C57BL/6 mouse was adopted as the standard even though the arterial lesions induced by even extreme diets were limited in size, complexity, a...
متن کاملA2b adenosine receptor regulates hyperlipidemia and atherosclerosis.
BACKGROUND The cAMP-elevating A(2b) adenosine receptor (A(2b)AR) controls inflammation via its expression in bone marrow cells. METHODS AND RESULTS Atherosclerosis induced by a high-fat diet in apolipoprotein E-deficient mice was more pronounced in the absence of the A(2b)AR. Bone marrow transplantation experiments indicated that A(2b)AR bone marrow cell signals alone were not sufficient to e...
متن کاملSex Difference and Postmenopausal Alteration of Atherosclerosis and Hyperlipidemia
It has been shown that the incidence of atherosclerotic disease such as coronary heart disease and stroke is lower in younger women than in men of the same age but increases after menopause, implying the athero-protective action of endogenous estrogen. In addition, observational studies have suggested the extensive efficacy of hormone replacement therapy (HRT) in postmenopausal women. The resul...
متن کاملVascular Medicine A2b Adenosine Receptor Regulates Hyperlipidemia and Atherosclerosis
Background—The cAMP-elevating A 2b adenosine receptor (A 2b AR) controls inflammation via its expression in bone marrow cells. Methods and Results—Atherosclerosis induced by a high-fat diet in apolipoprotein E– deficient mice was more pronounced in the absence of the A 2b AR. Bone marrow transplantation experiments indicated that A 2b AR bone marrow cell signals alone were not sufficient to eli...
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ژورنال
عنوان ژورنال: Japanese journal of MHTS
سال: 1998
ISSN: 1884-409X,0911-1840
DOI: 10.7143/jhep1985.25.300